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개최기간학술회의명개최장소발표제목
2011.2.26-2.28 2011 International Conference on Bioscience, Biochemistry and Bioinformatics Singapore Levosulpiride, (S)-(-)-5-Aminosulfonyl-N-[(1-Ethyl-2-Pyrrolidinyl) Methyl]-2-
2011.2.26-2.28 2011 International Conference on Bioscience, Biochemistry and Bioinformatics Singapore Protective Effects of Enhanced Tat-catalase Protein in Ischemic Insult by Enhancement of Transduction Efficiency
2011.2.26-2.28 2011 International Conference on Bioscience, Biochemistry and Bioinformatics Singapore Transduced Annexin І Protein Attenuate the Inflammatory Response in Vitro and in Vivo
2011.2.26-2.28 2011 International Conference on Bioscience, Biochemistry and Bioinformatics Singapore Effects of Tat-DJ-1 Protein on Oxidative Stress-Induced Neuronal Cell Death in Parkinson Disease Mouse Model
2010.12.18-21 2010 IEEE International Conference on Bioinformatics and Biomedicine Workshops Hong Kong PEP-1-Peroredoxin protein efficiently protects Raw 264.7 cells from lipopolysaccaride (LPS)-induced inflammation
2010.12.18-21 2010 IEEE International Conference on Bioinformatics and Biomedicine Workshops Hong Kong Enhancses the transduction efficiency of Tat-Catalase protein attenuate neuronal cell damage
2010.12.18-21 2010 IEEE International Conference on Bioinformatics and Biomedicine Workshops Hong Kong Transduced Tat-DJ-1 protein protects against oxidative stress-induced SH-SY5Y cells death and Parkinson disease mouse model
2010. 3.8-3.11 7th World Meeting on Pharmaceutics, Biopharmaceutics and Pharmaceutical Technology Malta Transduced DJ-1 fusion protein protects against dopaminergic neurons in Parkinson's Disease model
2010. 3.8-3.11 7th World Meeting on Pharmaceutics, Biopharmaceutics and Pharmaceutical Technology Malta Neuroprotective effects of PEP-1-Frataxin fusion protein after trasient forebrain ischemia in gerbils
2010. 3.8-3.11 7th World Meeting on Pharmaceutics, Biopharmaceutics and Pharmaceutical Technology Malta Transduced human PEP-1-FK506BP inhibits inflammatory cytokine expression in vitro and in vivo by supressing NF-kB activation
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